Today’s Morning Report is presented by Dr. Freedman!
The Case:
Left Ventricular Hypertrophy
Definition:
- increased mass of the left ventricle
How?:
- Things that cause increased afterload
HTN
Aortic Stenosis
Coarctation
- Genetics
HCOM
- Volume Overload?
Mitral Regurg
Aortic Regurg
Why do we care?
- risk of development of CAD
- risk of death
- Kannel et al, 1970
- ECG findings are characteristic and confusing
ECG findings of LVH
Basics:
- Thick-walled LV causes increased R wave amplitude in left-sided leads.
- Conversely, deep S waves in right-sided leads
- Thick-walls cause prolonged depolarization (increased time to peak of R) and abnormal repolarization (strain pattern?)
Voltage Criteria:
- There’s a billion
- Amal Mattu condenses them into
- S wave in V1 + R wave in V5-6 > 35mm
- Any S + any R > 45mm
- An R wave > 11mm in AVL
Non-Voltage Criteria
- Time to R wave peak > 50ms
- ST depressions and T wave inversions in lateral leads
- Characteristic morphology: ST depressions are diagonal and down-sloping
- Inverted T waves are very assymetric
NB* ECG criteria for LVH are only 50% sensitive!
Other findings commonly associated with LVH
- LAE
- LAD
- STE in right precordium
- U waves?!?!
LVH causes STE and ST Depressions!?!?!
How to distinguish LVH from STEMI?
- It’s not easy.
- Use caution
- Look out for horizontal ST depressions and symmetric inverted T waves.
When in doubt, get old and serial ecgs!
Have a low threshold to call cards.
Acknowledgements:
- http://ekgumem.tumblr.com/
- http://lifeinthefastlane.com/
- WILLIAM B. KANNEL, TAVIA GORDON, WILLIAM P. CASTELLI, JAMES R. MARGOLIS; Electrocardiographic Left Ventricular Hypertrophy and Risk of Coronary Heart DiseaseThe Framingham Study. Annals of Internal Medicine. 1970 Jun;72(6):813-822.
Jay Khadpe MD
- Editor in Chief of "The Original Kings of County"
- Assistant Professor of Emergency Medicine
- Assistant Residency Director
- SUNY Downstate / Kings County Hospital
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